[Bird populations--hatching grounds of pandemic influenza viruses?].
Identifieur interne : 000625 ( Main/Exploration ); précédent : 000624; suivant : 000626[Bird populations--hatching grounds of pandemic influenza viruses?].
Auteurs : Timm Harder [Allemagne] ; Christian Grund ; Martin Beer ; Christoph Staubach ; Claudia Schoene ; Hendrik Wilking ; Franz J. ConrathsSource :
- Berliner und Munchener tierarztliche Wochenschrift [ 0005-9366 ]
Descripteurs français
- KwdFr :
- Allemagne (épidémiologie), Animaux, Flambées de maladies (médecine vétérinaire), Grippe chez les oiseaux (transmission), Grippe chez les oiseaux (virologie), Grippe chez les oiseaux (épidémiologie), Grippe humaine (transmission), Grippe humaine (virologie), Grippe humaine (épidémiologie), Génome viral, Humains, Oiseaux (virologie), Orthomyxoviridae (pathogénicité), Réservoirs d'agents pathogènes (virologie), Sous-type H5N1 du virus de la grippe A (génétique), Sous-type H5N1 du virus de la grippe A (pathogénicité), Volaille (virologie), Évolution biologique.
- MESH :
- génétique : Sous-type H5N1 du virus de la grippe A.
- médecine vétérinaire : Flambées de maladies.
- pathogénicité : Orthomyxoviridae, Sous-type H5N1 du virus de la grippe A.
- virologie : Grippe chez les oiseaux, Grippe humaine, Oiseaux, Réservoirs d'agents pathogènes, Volaille.
- épidémiologie : Allemagne, Grippe chez les oiseaux, Grippe humaine.
- Animaux, Génome viral, Humains, Évolution biologique.
- Wicri :
- geographic : Allemagne.
English descriptors
- KwdEn :
- Animals, Biological Evolution, Birds (virology), Disease Outbreaks (veterinary), Disease Reservoirs (virology), Genome, Viral, Germany (epidemiology), Humans, Influenza A Virus, H5N1 Subtype (genetics), Influenza A Virus, H5N1 Subtype (pathogenicity), Influenza in Birds (epidemiology), Influenza in Birds (transmission), Influenza in Birds (virology), Influenza, Human (epidemiology), Influenza, Human (transmission), Influenza, Human (virology), Orthomyxoviridae (pathogenicity), Poultry (virology).
- MESH :
- geographic , epidemiology : Germany.
- epidemiology : Influenza in Birds, Influenza, Human.
- genetics : Influenza A Virus, H5N1 Subtype.
- pathogenicity : Influenza A Virus, H5N1 Subtype, Orthomyxoviridae.
- transmission : Influenza in Birds, Influenza, Human.
- veterinary : Disease Outbreaks.
- virology : Birds, Disease Reservoirs, Influenza in Birds, Influenza, Human, Poultry.
- Animals, Biological Evolution, Genome, Viral, Humans.
Abstract
Aquatic wild birds constitute the natural reservoir of influenza A viruses. Under appropriate selection pressure these viruses display a remarkable genetic flexibility which is based on their high mutation rate. At least 16 subtypes of the viral hemagglutinin glycoprotein (HA) und 9 of the viral neuraminidase (NA) are distinguishable as a result of natural evolution. Due to the segmentation of the viral genome, genetic reassortments and, thus, a theoretically unrestricted combination of HA and NA subtypes is possible. Transspecies-specific transmissions of viruses from the natural reservoirs to mammals (horses, swine, humans) led to the establishment of several influenza virus lineages which are circulating stably and independently from the previous reservoir. Introduction of viruses featuring new HA/NA combinations into immunologically naive populations can cause rapid and widespread, even pandemic, dissemination of such viruses. The (highly pathogenic avian influenza, HPAI). These mutants constitute the causative agents of classical fowl plague. HPAI viruses of subtypes H5 and H7 generally share a polybasic endoproteolytical cleavage site in the HA protein which allows for systemic replication in the avian host. Subtype H5 and H7 viruses which are maintained in the natural reservoir hosts, in contrast, are of low pathogenicity (LPAI) and harbour monobasic HA cleavage sites restricting their tissue tropism to epithelia of the respiratory and gastronintestinal tracts. This context forms the basis of all legal measures targeting the monitoring, prevention and eradication of HPAIV and LPAIV of subtypes H5 and H7 in poultry. Results of such measures in Germany in 2008 are presented and discussed.
PubMed: 19999379
Affiliations:
Links toward previous steps (curation, corpus...)
Le document en format XML
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Under appropriate selection pressure these viruses display a remarkable genetic flexibility which is based on their high mutation rate. At least 16 subtypes of the viral hemagglutinin glycoprotein (HA) und 9 of the viral neuraminidase (NA) are distinguishable as a result of natural evolution. Due to the segmentation of the viral genome, genetic reassortments and, thus, a theoretically unrestricted combination of HA and NA subtypes is possible. Transspecies-specific transmissions of viruses from the natural reservoirs to mammals (horses, swine, humans) led to the establishment of several influenza virus lineages which are circulating stably and independently from the previous reservoir. Introduction of viruses featuring new HA/NA combinations into immunologically naive populations can cause rapid and widespread, even pandemic, dissemination of such viruses. The (highly pathogenic avian influenza, HPAI). These mutants constitute the causative agents of classical fowl plague. HPAI viruses of subtypes H5 and H7 generally share a polybasic endoproteolytical cleavage site in the HA protein which allows for systemic replication in the avian host. Subtype H5 and H7 viruses which are maintained in the natural reservoir hosts, in contrast, are of low pathogenicity (LPAI) and harbour monobasic HA cleavage sites restricting their tissue tropism to epithelia of the respiratory and gastronintestinal tracts. This context forms the basis of all legal measures targeting the monitoring, prevention and eradication of HPAIV and LPAIV of subtypes H5 and H7 in poultry. Results of such measures in Germany in 2008 are presented and discussed.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">19999379</PMID><DateCompleted><Year>2010</Year><Month>04</Month><Day>29</Day></DateCompleted><DateRevised><Year>2010</Year><Month>11</Month><Day>18</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0005-9366</ISSN><JournalIssue CitedMedium="Print"><Volume>122</Volume><Issue>11-12</Issue><PubDate><MedlineDate>2009 Nov-Dec</MedlineDate></PubDate></JournalIssue><Title>Berliner und Munchener tierarztliche Wochenschrift</Title><ISOAbbreviation>Berl. Munch. Tierarztl. Wochenschr.</ISOAbbreviation></Journal><ArticleTitle>[Bird populations--hatching grounds of pandemic influenza viruses?].</ArticleTitle><Pagination><MedlinePgn>440-5</MedlinePgn></Pagination><Abstract><AbstractText>Aquatic wild birds constitute the natural reservoir of influenza A viruses. Under appropriate selection pressure these viruses display a remarkable genetic flexibility which is based on their high mutation rate. At least 16 subtypes of the viral hemagglutinin glycoprotein (HA) und 9 of the viral neuraminidase (NA) are distinguishable as a result of natural evolution. Due to the segmentation of the viral genome, genetic reassortments and, thus, a theoretically unrestricted combination of HA and NA subtypes is possible. Transspecies-specific transmissions of viruses from the natural reservoirs to mammals (horses, swine, humans) led to the establishment of several influenza virus lineages which are circulating stably and independently from the previous reservoir. Introduction of viruses featuring new HA/NA combinations into immunologically naive populations can cause rapid and widespread, even pandemic, dissemination of such viruses. The (highly pathogenic avian influenza, HPAI). These mutants constitute the causative agents of classical fowl plague. HPAI viruses of subtypes H5 and H7 generally share a polybasic endoproteolytical cleavage site in the HA protein which allows for systemic replication in the avian host. Subtype H5 and H7 viruses which are maintained in the natural reservoir hosts, in contrast, are of low pathogenicity (LPAI) and harbour monobasic HA cleavage sites restricting their tissue tropism to epithelia of the respiratory and gastronintestinal tracts. This context forms the basis of all legal measures targeting the monitoring, prevention and eradication of HPAIV and LPAIV of subtypes H5 and H7 in poultry. Results of such measures in Germany in 2008 are presented and discussed.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Harder</LastName><ForeName>Timm</ForeName><Initials>T</Initials><AffiliationInfo><Affiliation>Institut für Virusdiagnostik, Friedrich-Loeffler Institut, Greifswald-Insel Riems. timm.harder@fli.bund.de</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Grund</LastName><ForeName>Christian</ForeName><Initials>C</Initials></Author><Author ValidYN="Y"><LastName>Beer</LastName><ForeName>Martin</ForeName><Initials>M</Initials></Author><Author ValidYN="Y"><LastName>Staubach</LastName><ForeName>Christoph</ForeName><Initials>C</Initials></Author><Author ValidYN="Y"><LastName>Schoene</LastName><ForeName>Claudia</ForeName><Initials>C</Initials></Author><Author ValidYN="Y"><LastName>Wilking</LastName><ForeName>Hendrik</ForeName><Initials>H</Initials></Author><Author ValidYN="Y"><LastName>Conraths</LastName><ForeName>Franz J</ForeName><Initials>FJ</Initials></Author><Author ValidYN="Y"><CollectiveName>EU Monitoringprogramm für AIV beteiligten Untersuchungseinrichtungen der Bundesländer</CollectiveName></Author></AuthorList><Language>ger</Language><PublicationTypeList><PublicationType UI="D004740">English Abstract</PublicationType><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D016454">Review</PublicationType></PublicationTypeList><VernacularTitle>Vogelpopulationen--Brutstätten pandemischer Influenzaviren?</VernacularTitle></Article><MedlineJournalInfo><Country>Germany</Country><MedlineTA>Berl Munch Tierarztl Wochenschr</MedlineTA><NlmUniqueID>0003163</NlmUniqueID><ISSNLinking>0005-9366</ISSNLinking></MedlineJournalInfo><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D000818" MajorTopicYN="N">Animals</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005075" MajorTopicYN="N">Biological Evolution</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D001717" MajorTopicYN="N">Birds</DescriptorName><QualifierName UI="Q000821" MajorTopicYN="N">virology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D004196" MajorTopicYN="N">Disease Outbreaks</DescriptorName><QualifierName UI="Q000662" MajorTopicYN="N">veterinary</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D004197" MajorTopicYN="N">Disease Reservoirs</DescriptorName><QualifierName UI="Q000821" MajorTopicYN="N">virology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D016679" MajorTopicYN="N">Genome, Viral</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005858" MajorTopicYN="N" Type="Geographic">Germany</DescriptorName><QualifierName UI="Q000453" MajorTopicYN="N">epidemiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D053124" MajorTopicYN="N">Influenza A Virus, H5N1 Subtype</DescriptorName><QualifierName UI="Q000235" MajorTopicYN="N">genetics</QualifierName><QualifierName UI="Q000472" MajorTopicYN="Y">pathogenicity</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D005585" MajorTopicYN="N">Influenza in Birds</DescriptorName><QualifierName UI="Q000453" MajorTopicYN="N">epidemiology</QualifierName><QualifierName UI="Q000635" MajorTopicYN="N">transmission</QualifierName><QualifierName UI="Q000821" MajorTopicYN="N">virology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D007251" MajorTopicYN="N">Influenza, Human</DescriptorName><QualifierName UI="Q000453" MajorTopicYN="Y">epidemiology</QualifierName><QualifierName UI="Q000635" MajorTopicYN="N">transmission</QualifierName><QualifierName UI="Q000821" MajorTopicYN="N">virology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D009975" MajorTopicYN="N">Orthomyxoviridae</DescriptorName><QualifierName UI="Q000472" MajorTopicYN="N">pathogenicity</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D011200" MajorTopicYN="N">Poultry</DescriptorName><QualifierName UI="Q000821" MajorTopicYN="N">virology</QualifierName></MeshHeading></MeshHeadingList><NumberOfReferences>26</NumberOfReferences></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="entrez"><Year>2009</Year><Month>12</Month><Day>17</Day><Hour>6</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="pubmed"><Year>2009</Year><Month>12</Month><Day>17</Day><Hour>6</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2010</Year><Month>4</Month><Day>30</Day><Hour>6</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">19999379</ArticleId></ArticleIdList></PubmedData></pubmed><affiliations><list><country><li>Allemagne</li></country></list><tree><noCountry><name sortKey="Beer, Martin" sort="Beer, Martin" uniqKey="Beer M" first="Martin" last="Beer">Martin Beer</name><name sortKey="Conraths, Franz J" sort="Conraths, Franz J" uniqKey="Conraths F" first="Franz J" last="Conraths">Franz J. Conraths</name><name sortKey="Grund, Christian" sort="Grund, Christian" uniqKey="Grund C" first="Christian" last="Grund">Christian Grund</name><name sortKey="Schoene, Claudia" sort="Schoene, Claudia" uniqKey="Schoene C" first="Claudia" last="Schoene">Claudia Schoene</name><name sortKey="Staubach, Christoph" sort="Staubach, Christoph" uniqKey="Staubach C" first="Christoph" last="Staubach">Christoph Staubach</name><name sortKey="Wilking, Hendrik" sort="Wilking, Hendrik" uniqKey="Wilking H" first="Hendrik" last="Wilking">Hendrik Wilking</name></noCountry><country name="Allemagne"><noRegion><name sortKey="Harder, Timm" sort="Harder, Timm" uniqKey="Harder T" first="Timm" last="Harder">Timm Harder</name></noRegion></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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